By Icon Health Publications
It is a 3-in-1 reference e-book. It provides an entire clinical dictionary protecting hundreds and hundreds of phrases and expressions on the subject of arteriosclerosis. It additionally offers broad lists of bibliographic citations. eventually, it offers details to clients on how you can replace their wisdom utilizing quite a few net assets. The publication is designed for physicians, scientific scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to get to grips with study devoted to arteriosclerosis. in the event that your time is efficacious, this e-book is for you. First, you won't waste time looking the web whereas lacking loads of proper info. moment, the ebook additionally saves you time indexing and defining entries. ultimately, you won't waste time and cash printing hundreds and hundreds of websites.
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Extra resources for Arteriosclerosis - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
The specific aims are to: (i) define, by comprehensive qualitative and quantitative analysis, the effects of ICAM-1 deficiency on development and progression, organ distribution, and inflammatory characteristics of lesions of vasculitis in MRL/MpJ-Faslpr mice; (ii) define the relative contributions of ICAM-1 in mediating neutrophil, lymphocyte, and monocyte adhesion to MRL/MpJ-Faslpr endothelial cells; (iii) determine the roles of ICAM-1 in neutrophilmediated damage to MRL/MpJ-Faslpr endothelial cells; and (iv) determine whether 28 Arteriosclerosis MRL/MpJ-Faslpr mice with null mutations in the ICAM-1 ligands CD11a (LFA-1), CD11b (Mac-1), or CD11c (p150/95) will alter development and progression, organ distribution, or inflammatory characteristics of lesions of vasculitis.
Heparin and dermatan sulfate accelerate its rate of inhibition 10 to 10-fold through an allosteric mechanism that is only partly understood. This unique mechanism appears to involve binding of the N-terminus of HCII to exosite I of thrombin following the binding of glycosaminoglycans (GAG). Although some of the residues involved in this mechanism have been identified through site-directed mutagenesis, the structural changes that occur have not been identified. Another unusual feature of HCII is its active site sequence, which makes the serpin more sensitive to activation by particular GAG and therefore confers greater site specificity.
This work will provide considerable insight into the role of these disparate signaling modalities in the angiogenic response. ; Assistant Professor; Genetics; University of Alabama at Birmingham Uab Station Birmingham, Al 35294 Timing: Fiscal Year 2002; Project Start 01-APR-2002; Project End 31-MAR-2007 Summary: Systemic lupus erythematosus, Wegener's granulomatosis, and polyarteritis nodosa are chronic inflammatory diseases that have vasculitis as a major component. The pathogenesis of vasculitis in these and other disorders involves a complex interaction among inflammatory, genetic, and environmental factors.